By Renate Unsöld, Wolfgang Seeger (auth.)
This complete monograph opens up sensational new diagnostic and healing views. The topographic details is gifted with first-class anatomic arrangements. The vast spectrum of indicators is taken from vast medical adventure; they're significantly analysed and in comparison to the ophthalmological, neurosurgical, and neuroradiological literature. The monograph is a wonderful resource for the ophthalmologic and neurologic clinician who's the 1st to be faced with indicators of optic nerve lesions. For the radiologist, it bargains a transparent, didactic assessment of commonplace pathological alterations of crucial lesions. For the neurosurgeon, the dialogue of optimum strategy and intraoperative findings issues to the potential for early microsurgical intervention that keeps as a lot functionality as attainable.
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Additional resources for Compressive Optic Nerve Lesions at the Optic Canal: Pathogenesis — Diagnosis — Treatment
27. Pre- and postoperative YEP in a patient with a surgically proven right optic nerve compression due to dolichoectatic internal carotid artery and a left pneumosinus dilatans causing narrowing of the left optic canal (Case 2). Above: preoperative YEP. For both eyes, latency is above normal limits, amplitude is below normal limits. Below: postoperative VEP. Right eye (RE): latency slightly above normal limits, amplitude low but within normal limits. Conclusion: Preoperatively, the marked reduction of amplitude combined with a moderate latency increase points to blockage of conduction by compression rather than to an inflammatory disease.
3) appear to be sufficient indication for microsurgical decompres36 sion. The typical clinical, radiological, and intraoperative findings in optic nerve compression by ectatic vessels as well as the functional results of decompressive surgery are demonstrated in cases 1-4 of the selected case reports. These findings indicate not only the existence of this disease entity but also that decompression by microsurgical techniques may not only preserve but also improve visual function. Sphenoid si nu ' Optic canals ( narrowed) Intern al carotid artery (dol ichoectatic) Intern al ca rotid artery (do lichocctatic) O pt ic chia 'm (prefixed ) Fig.
Before computerized tomography was introduced, the condition could not be demonstrated in the living patient. Neither is there a correlation between calcifications of the carotid artery, as seen in conventional radiography, nor of the lumen of the arteries demonstrated by angiography (Knapp 1940; Saphir 1933; Walsh and Hoyt 1969). The vascular changes responsible for nerve compression in the absence of gross calcifications or stenosis lie within the vessel wall, which could not be visualized prior to thin section CT.
Compressive Optic Nerve Lesions at the Optic Canal: Pathogenesis — Diagnosis — Treatment by Renate Unsöld, Wolfgang Seeger (auth.)