By Christie M. Ballantyne MD
Dr. Ballantyne-one of the most important lipid specialists on this planet and recruited by means of Dr. Braunwald's middle sickness editorial team-together with a stellar forged of participants offers the entire clinical and scientific details you must successfully deal with each element of dyslipidemia. From simple technological know-how to pathogenesis of atherothrombotic ailment to chance evaluation and the most recent remedy suggestions, this new name within the Braunwald's center affliction family members deals unprecedented up to date insurance and specialist advice on lipidology in a simple, obtainable, and ordinary variety. Plus, specialist seek advice performance supplies each time, anyplace entry to the entire textual content on-line. . contains complete textual content on-line entry at expertconsult.com. . gains the services of 1 of the key specialists within the box, making sure you get authoritative suggestions with the main definitive wisdom on hand. . comprises wide clinically suitable info masking probability evaluate, remedy, distinct sufferer populations, and experimental cures, together with focusing on HDL that can assist you successfully deal with any demanding situations you face. . makes use of therapy algorithms for simple entry to key content material. . offers present perform instructions that help in the decision-making technique.
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Extra resources for Clinical Lipidology: A Companion to Braunwald's Heart Disease
Myant NB, Forbes SA, Day IN, Gallagher J: Estimation of the age of the ancestral arginine3500→glutamine mutation in human apoB-100. Genomics 1997;45:78–87. 35. ] Atherosclerosis 1993;104:1–18. 36. Schaefer JR, Scharnagl H, Baumstark MW, et al: Homozygous familial defective apolipoprotein B-100. Enhanced removal of apolipoprotein E-containing VLDLs and decreased production of LDLs. Arterioscler Thromb Vasc Biol 1997;17:348–353. 37. Linton MF, Farese RV Jr, Young SG: Familial hypobetalipoproteinemia.
As a result, inhibition or loss of some of the pancreatic lipolytic enzyme activities would be unlikely to result in an appreciable reduction of cholesterol absorption. 11 The regulatory effects of the group 1B phospholipase A2, as well as pancreatic lipase-related protein–1 and –2 on intestinal cholesterol absorption, have not yet been deﬁned. The digestion of triglycerides also begins in the stomach. The key enzymes are lingual lipase secreted by the salivary gland and gastric lipase secreted by the gastric mucosa.
136. Koster A, Chao YB, Mosior M, et al: Transgenic angiopoietinlike (angptl)4 overexpression and targeted disruption of angptl4 and angptl3: regulation of triglyceride metabolism. Endocrinology 2005;146:4943–4950. 150. Heeren J, Weber W, Beisiegel U: Intracellular processing of 25 endocytosed triglyceride-rich lipoproteins comprises both recycling and degradation. J Cell Sci 1999;112(Pt 3):349–359. 151. Swift LL, Farkas MH, Major AS, et al: A recycling pathway for resecretion of internalized apolipoprotein E in liver cells.
Clinical Lipidology: A Companion to Braunwald's Heart Disease by Christie M. Ballantyne MD