By Hans Hoff, Kurt Jellinger (auth.), Igor Klatzo, Franz Seitelberger (eds.)
The Symposium on mind Edema has been equipped through the Osterreichische Arbeitsgemeinschaft fur Neuropathologie and via the matter fee for Neuropathology of the realm Federa tion of Neurology. we wish to precise our because of Dr. L. van Bogaert, president of the W.F.N., for his nice curiosity within the layout of this convention and for his vigorous help. one of many goals ofthe challenge fee for Neuropathology has been to give a contribution to the advances in a number of difficulties that are changing into ripe for elucidation by means of specialists in several fields. the assumption of the Symposium on mind Edema has as its history a compelling necessity to clarify this topic that's clinically so very important and neuropathologic best friend so poorly understood. It was once was hoping that this is able to be accomplished via a multidisciplinary strategy regarding various facets to be mentioned and correlated. mind edema, being an irregular cerebral situation, belongs primarily to the sector of neuropathology, yet to achieve success in our endeavors we needed to follow neuropathology in its most recent and widest phrases. via this we take into account that such neuropathology contains the examine of all irregular phenomena within the worried tissue on the topic of the structural changes, and via "structural" we suggest not just tissual or mobile ones but additionally adjustments on ultrastructural and molecular degrees. therefore we wanted shut and built-in coopera tion of specialists in fields corresponding to neurohistology, histochemistry, electron microscopy, neurochemistry, and neurophysiology.
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Extra resources for Brain Edema: Proceedings of the Symposium September 11–13, 1965, Vienna
From our knowledge of disturbances of water and ion metabolism, a serious warning must be uttered against any blind, rule-of-thumb administration of dehydrating substances. Any physico- chemically induced withdrawal of fluid and electrolytes has to be based on a stable and compensated circulatory system. Dehydration therapy in each individual case has to be controlled and regulated following the schedule of equilibrated metabolic balances. Any indiscriminate use of such methods involves the risk of setting up undesirable counter- regulatory mechanisms.
Today we are able to treat increase of brain volume successfully up to certain stages of its development, since we know something about its nature and course. Moreover, we are acquainted with symptoms that may announce the onset of cerebral edema and its dangerous development associated with increase of intracranial pressure. The danger that medical help is too late may occur, however, when the clinical syndrome of BE is already well advanced, when the brain is more or less saturated with fluid or irreversible imbalances of water and ion transport are manifest.
Weinstein and N. F. Kassel: Cerebral vasomotor paralysis produced by intracranial hypertension, Trans. Amer. Neurol. , 214- 215 (1964). Lazorthes, G. and L. Campan: L'oedeme cerebral. Paris: Masson et Cie. (1963). Lee, J. C. and L. Bakay: Ultrastructural changes in the edematous central nervous system, 1: Triethyltin edema, Arch. Neurol. (Chicago), 13,48-57 (1965). Levy, W. , J. M. Taylor, I. Herzog and L. C. Scheinberg: The effect of hypertonic urea on cerebral edema in the rabbit induced by triethyl tin sulfate, Arch.
Brain Edema: Proceedings of the Symposium September 11–13, 1965, Vienna by Hans Hoff, Kurt Jellinger (auth.), Igor Klatzo, Franz Seitelberger (eds.)