By Philip M. Parker
In March 2001, the nationwide Institutes of healthiness issued the subsequent caution: "The variety of sites supplying health-related assets grows each day. Many websites supply precious info, whereas others can have info that's unreliable or misleading." moreover, a result of fast raise in Internet-based info, many hours should be wasted looking out, identifying, and printing. for the reason that basically the smallest fraction of knowledge facing Alzheimer disorder is listed in se's, comparable to www.google.com or others, a non-systematic method of web learn should be not just time eating, but additionally incomplete. This publication used to be created for doctors, scholars, and contributors of most people who are looking to behavior scientific study utilizing the main complex instruments to be had and spending the smallest amount of time doing so.
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Additional resources for Alzheimer Disease - A Bibliography and Dictionary for Physicians, Patients, and Genome Researchers
The results of numerous published neuropathological studies suggest that the hippocampal formation is the initial locus in the disease processes of AD. In addition, the progression of neurodegenerative changes is remarkably uniform across individuals, is predictable, and shows little inter-patient variation. In AD, the lesions eventually lead to severe damage to the hippocampus (referred to as the "floor effect") and clinical expression of dementia. Our preliminary results demonstrate that the COSLOF index has the ability to distinguish between probable/possible AD patients and cognitively healthy controls.
At death a comprehensive neuropathologic examination will be performed and the clinical, radiological and pathological views of the disease correlated. Tissues and DNA obtained from these subjects will be available for research on the biology and genetics of the disease. Individual research projects within the ADRC will examine various aspects of the cellular and molecular biology of AD as well as an in-depth analysis of brain function in 28 Alzheimer Disease human subjects using MRI. Patients enrolled in the ADRC will have the opportunity to participate in trials of new drugs and treatments for dementing diseases as they become available.
In the third aim, we will use cortical neurons cultured from neonatal transgenic mice to determine at the cell level the mechanisms by which antioxidant vitamins lessen lipid peroxidative damage. If antioxidant vitamins ameliorate toxicity in cell and animal models of Alzheimer's disease, then oxidant stress is involved in disease progression, and antioxidant vitamin supplements may be beneficial. ; Neurology and Neuroscience; Weill Medical College of Cornell Univ 1300 York Avenue New York, Ny 10021 Timing: Fiscal Year 2006; Project Start 30-SEP-2006; Project End 31-JUL-2011 Summary: (provided by applicant): Beta-amyloid is a major constituent of the plaques found in the brains of patients afflicted by Alzheimer's disease and reduction in beta- Studies 33 amyloid is currently a major target of therapeutic strategies for the disease.
Alzheimer Disease - A Bibliography and Dictionary for Physicians, Patients, and Genome Researchers by Philip M. Parker