By Bernard S. Kaplan M.B., B.Ch., F.C.P.(S.A.), Paul R. Goodyer M.D. (auth.), José Strauss MD (eds.)
My ideas in regards to the Hemolytic Uremic Syndrome (HUS) acquired began in 1961 in addition to my try to go back to Argentina. As I sought my method in Buenos Aires, I visited Carlos Gianantonio whom I had met in Caracas the yr ahead of in the course of the Pan American pediatric conferences. at the moment he used to be actively engaged on HUS which had turn into a virus in Buenos Aires and different components of Argentina. i used to be inspired through the workforce attempt and devotion of his staff to such heavy calls for. They evidently have been assembly the problem at an amazingly excessive point less than a truly crippling actual scenario with shortages of house, laboratories and gear. His crew including Dr. Becu, on the time the pathologist on the kid's sanatorium of Buenos Aires (we had met via his mom who used to be instrumental in arranging my go back to Buenos Aires), wrote a few of the vintage papers on HUS. over the years as Dr. Gianantonio turned extra enthusiastic about normal pediatrics, the executive elements and its orientation in Latin the United States, he turned identified for his deep philosophical questions as to what we're doing and the place we're going. His questions have visible implications relating to an agressive method of our pediatric nephrology patients.
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Extra info for Acute Renal Disorders and Renal Emergencies: Proceedings of Pediatric Nephrology Seminar X held at Bal Harbour, Florida, January 30 – February 3, 1983
The rate of fall in GFR, in addition to reflecting the intrinsic insult to the renal vascular endothelium, may be hastened by concomitant dehydration (15), intraluminal uric acid deposi tion (18, 19), and changes in circulating levels of catecholamine and angiotensin II. (20) Another renal rrechanism may be related to the effect of volume depletion on net uric acid excretion. Many children with HUS experience sane degree of volume depletion because of losses of gastrointestinal contents and anorexia.
Scans obtained in the sickest infants, suggests that the pathogenesis of the central nervous system injury may not be different from that of the renal damage. As in other types of acute renal failure, in HUS, the heart may present many functional abnormalities related to metabolic and hemodynamic variables. The presence of multiple small infarcts importance has not been adequately stressed. that attain clinical As was mentioned before, the myocardial lesion was a determinant of death in some patients either because of cardiac failure or arrythmia.
83: 271, 1973. 9. : The treatment of the hemolytic-uremic syndrome with inhibitors of platelet function. Am. J. , 66: 711, 1979. 10. : streptokinase and anti-thrombotic therapy in the hemolytic-uremic syndrome. J. , 84: 345, 1974. 11. : Thrombolytic therapy in haemolytic-uraemic syndrome. Br. Med. , 3: 217, 1974. 12. : hemolytic-uremic syndrome with plasma. Treatment of the Clin. , 12: 279, 1979. 13. : Haemolytic-uraemic syndrome: Deficiency of plasma factor(s) regulating prostacyclin activity?
Acute Renal Disorders and Renal Emergencies: Proceedings of Pediatric Nephrology Seminar X held at Bal Harbour, Florida, January 30 – February 3, 1983 by Bernard S. Kaplan M.B., B.Ch., F.C.P.(S.A.), Paul R. Goodyer M.D. (auth.), José Strauss MD (eds.)